https://nova.newcastle.edu.au/vital/access/ /manager/Index ${session.getAttribute("locale")} 5 https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:36160 Thu 09 Dec 2021 11:04:05 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:8399 Sat 24 Mar 2018 08:40:59 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:16009 Sat 24 Mar 2018 08:19:29 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:12458 Sat 24 Mar 2018 08:17:49 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:11067 Sat 24 Mar 2018 08:13:32 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:21419 P=0.02), diastolic (P=0.01), and arterial blood pressures (P=0.002), respectively, 5 years later. In girls, each 1-SD increase in dietary glycemic index, glycemic load, carbohydrate, and fructose was concurrently related to increases of 1.81 (P=0.001), 4.02 (P=0.01), 4.74 (P=0.01), and 1.80 mm Hg (P=0.03) in systolic blood pressure, respectively, >5 years. Significant associations between carbohydrate nutrition variables and blood pressure were not observed among boys. Excessive dietary intake of carbohydrates, specifically from high glycemic index/glycemic load foods, could adversely influence blood pressure, particularly in girls, whereas fiber-rich diets may be protective against elevated blood pressure during adolescence.]]> Sat 24 Mar 2018 08:05:02 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:18198 Sat 24 Mar 2018 08:04:50 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:19646 p < 0.001) trials compared to 120 and 180 min following ingestion for the 6 and 9 mg kg⁻¹ fed trials, respectively (p < 0.001). Peak concentration was greater in the 9 mg kg⁻¹ fasted trial than the corresponding fed condition (70 ± 9 μmol L⁻¹ and 56 ± 6 μmol L⁻¹, respectively) and both were greater than the 6 mg kg⁻¹ conditions (44 ± 8 μmol L⁻¹ and 38 ± 8 μmol L⁻¹ for 6 mg kg⁻¹ fasted and fed, respectively). Area under the caffeine curve was significantly greater (p < 0.001) in the 9 mg kg⁻¹ fasted trial (3262 μmol L⁻¹ h⁻¹), whilst areas were lowest in the 6 mg kg⁻¹ fed trial (1644 μmol L⁻¹ h⁻¹). Conclusions: A high carbohydrate meal consumed prior to caffeine ingestion significantly reduced serum caffeine concentrations and delayed time to peak concentration. Differences in research findings between caffeine supplementation studies may, at least in part, be related to variations in postprandial timing of caffeine intake. The influence of postprandial timing should be considered when athletes consume caffeine with the aim of enhancing performance.]]> Sat 24 Mar 2018 08:01:09 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:16926 Sat 24 Mar 2018 08:00:29 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:26704 Sat 24 Mar 2018 07:26:20 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:22661 Sat 24 Mar 2018 07:15:39 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:45562 P =0.001], with inter‐individual requirements ranging from 0.9 to six times the low‐protein meal requirement. Approximately half the additional insulin [1.1 units/h (CI 0.5, 1.8; P =0.001)] was given in the first 2 h, compared with an additional 0.5 units/h (CI –0.2, 1.2; P =0.148) in the second 2 h and 0.1 units (CI –0.6, 0.8; P =0.769) in the final hour. Conclusions: A high‐protein meal requires ~50% more insulin to maintain euglycaemia than a low‐protein meal that contains the same quantity of carbohydrate. The majority is required within the first 2 h. Inter‐individual differences exist in insulin requirements for dietary protein.]]> Fri 04 Nov 2022 14:45:18 AEDT ]]>